Atopic Dermatitis (Eczema)
Type I (IgE) hypersensitivity inflammatory reaction 
Risk factors: Family history of atopy.   Exacerbated by scratching, stress
Epidemiology: Usually begins prior to 6m of age. 
(FACE): flexor surfaces get adults, children extensor)

Acute form
Pruritus. 
Appear erythematous, edematous with papules/plaques.
Scaling, weeping, and crusting  

Chronic form
Lichenification
painful fissures

Contact Dermatitis
Cell mediated reaction involving sensitized T lymphocytes. 
Etiology 
Irritant form: Chemical insult to skin. No previous sensitizing event. 
Allergic form is delayed-hypersensitivity reaction. Skin sensitized from initial exposure. During next exposure patient has reaction. 
Develop 24-96h post exposure
Pruritus
Acute present as vesicles with clear fluid on erythematous  edematous skin. 
Sub-acute is edema and papules

Stasis Dermatitis
Inflammatory skin disease that occurs on  lower extremities 
Extravasation of plasma proteins and RBC into subcutaneous tissues. Becomes brown in color due to hemosiderin deposits 
Results in interstitial fluid accumulation . Leads to reduced capillary blood flow 
Can progress to venous stasis ulcers and fibrosis
Found in 6-7% of elderly population

Diaper Dermatitis
Irritant dermatitis 
Cutaneous Candidiasis infection (C. Albicans )
Risks: areas where warmth and moisture lead to maceration of skin or mucous membranes 

Seborrheic Dermatitis
Skin rash that occurs in areas of high sebaceous gland concentration 
Cutaneous inflammation to dermis 
Etiology: Immune response to endogenous yeast Pityrosporum 
Triggered by seasonal changes, scratching, emotional stress, medications... 

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